Genetic obesity is a condition in which an individual is genetically predisposed to having a low body mass index (BMI).
People with genetic obesity are likely to have a BMI below 30, and have a risk of developing obesity-related conditions.
For example, some people with genetic type I obesity are more likely to develop type 2 diabetes.
Some people with genes for obesity also have genes for insulin resistance, which increases the risk of diabetes.
In order to be considered for treatment, people with a BMI of 30 or above have to be diagnosed with obesity-associated conditions, such as Type 2 diabetes, high blood pressure, or heart disease.
Some people also need a doctor’s prescription for weight loss drugs to avoid a high BMI.
Researchers from the University of Melbourne and Melbourne University Hospital have found that genetic obesity can be caused by a number of genes, and can be triggered by the stress of being overweight or obese.
It is now understood that genetics are involved in determining whether people develop obesity-specific conditions, and that these genetic differences can be passed on to offspring.
Genetic obesity is also associated with the development of insulin resistance and the development in the womb of the insulin-producing cells called adipocytes.
People with obesity and insulin resistance can have abnormal insulin production, and may have low levels of circulating levels of the hormone insulin.
In some cases, people who are obese also have insulin resistance.
People who have inherited the gene for genetic obesity also appear to have increased risk of obesity-Related disorders, such that people with the gene may have higher body mass indices, and a higher BMI.
Genetic obesity may also be a risk factor for type 2 Diabetes.
Obesity can also be associated with an increased risk for type 1 Diabetes, which is a type of diabetes caused by abnormal insulin secretion.
Obesity and diabetes are linked to a range of health problems, such: weight gain, increased risk, and obesity-induced insulin resistance are all risk factors for the development and progression of diabetes in both adults and children.